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In cells depleted of GSH, p16 expression was significantly decreased by rapamycin and increased by 3-MA and Baf-A1 (Fig

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Furthermore, mitochondrial biogenesis of T-cells can also be repressed by TME, leading to metabolic insufficiency and dysfunction, eventually compromising T-cell responses [180]

Table 2 Redox related proteins associated dysregulation in ALS

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