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demonstrated that the GLP-1 analogue lixisenatide decreases atherosclerosis in insulin-resistant Apoe/Irs2+/ mice by promoting macrophage polarization towards an anti-inflammatory M2 phenotype, which was associated with enhanced plaque stability (i.e., thicker fibrous caps, fewer necrotic cores, and decreased inflammatory infiltrates)

(6) This form may be particularly beneficial for those seeking quicker effects from their supplementation

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involvement in the metabolism of estrogens, leukotrienes, and prostaglandins Improves DNA synthesis

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