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Lack of oral GSH supplementation effects upon growth or changes in serum or fecal inflammatory markers in children with cystic fibrosis with pancreatic insufficiency (125) could be probably explained by the inability of the cells to uptake extracellular GSH to be used inside the cells
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In particular, the enhanced GSH system plays an essential role in the antioxidant capacity of RSV [191]
Oral administration is not viablegastric acid and hepatic first-pass metabolism degrade peptide bonds within minutes, rendering the compounds inactive before reaching systemic circulation
mitochondrial, SHMT2) catalyzes the transfer of the beta carbon of serine to tetrahydrofolate (THF) to form glycine and one-carbon units, 5,10-methylene-THF (Figure 1), which is critical for nucleotide synthesis
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