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137 These results bolster the notion that GLP-1 RAs could have an impact on cellular aging and potentially diminish age-related cellular aging in humans

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Chondrocytes were passaged at 8090% confluency

For instance, in cardiomyocytes, mitochondrial impairment may primarily disrupt energy production essential for cardiac contraction, while in neurons, it can lead to synaptic dysfunction and neurodegeneration [32]