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(1) This peptide appears to be naturally present in the bloodstream and has been suggested as a potential element in regulating the growth, development, and apoptosis of vascular endothelial cells

Only clinical study designs (S)randomised controlled trials (RCTs), non-RCTs, cohort, or case-control studieswere included

The specific myelin damage resulting from B12 deficiency has also been connected to B12 reactions related to MUT, which is needed to convert methylmalonyl coenzyme A into succinyl coenzyme A

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