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These changes collectively lead to adipose tissue dysfunction, characterized by increased release of inflammatory cytokines, enhanced lipolysis, impaired insulin signaling, and the development of leptin resistance, ultimately leading to the typical metabolic abnormal phenotype characteristic of PCOS (51, 5355)

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Further, we found a decrease in TKV in all active treatment groups, which was possibly mediated by a reduction in hyperfiltration

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No shortages here in Boston, Fitch said this week

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