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Acute chemogenetic silencing of NSM neurons suppressed aversion to the mediocre crp and cysE diets, recapitulating the effects of the tph-1 knockout in NSM (Fig

Mitochondrial complex III deficiency caused by a homozygous UQCRC2 mutation presenting with neonatal-onset recurrent metabolic decompensation

Hamilton NB, Attwell D, Hall CN

It acts as a major intracellular antioxidant, neutralizing free radicals and helping regenerate other antioxidants, such as vitamin C