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These involve protein synthesis, metabolism of toxins, and immune function

The initiation of intestinal immune imbalance in IBD is driven by a profound ecological shift away from microbial tolerance. Under healthy conditions, the microbiota maintains homeostasis through the production of short-chain fatty acids (SCFAs), particularly butyrate, which promote the differentiation of anti-inflammatory regulatory T cells (Treg) and suppress pro-inflammatory Th1 and Th17 cell maturation (Acevedo-Romn et al., 2024)

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NDI1 expression led to increased NAD + , nucleotides and -ketoglutarate with reduced succinate