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For instance, from the perspective of clinical trial research, Samulak et al

F-bio-HJ triggers starvation protection in macrophages via glucose oxidase to reduce cellular ferroptosis while accelerating Fe delivery to bacteria (both extracellularly and intracellularly) to induce bacterial ferroptosis

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Furthermore, many researchers have found that reduced GSH levels and GSH-dependent antioxidant enzyme activity in damaged myocardium due to excess ROS and disruption of the GSH synthesis process, along with increased oxidative damage and ferroptosis

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