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Glutaredoxin-2 is required to control oxidative phosphorylation in cardiac muscle by mediating deglutathionylation reactions

Overall, the mechanisms by which human OLs undergo cell death are still being resolved, with the potential for new regulated cell death mechanisms to be uncovered and the development of novel therapeutic interventions

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248 Also, SAM has a key role in regulating liver homeostasis, and reduced levels of SAM synthesis have been detected in various chronic liver injuries, such as NAFLD

This was confirmed by shRNA to GCLC, the target of BSO, which increased glutamate levels (Fig