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TSS pathway activity is suppressed by excess hemin under hypoxia, and the suppression is reversible through DMF treatment The afore findings suggest that supplementing cystine at physiological concentrations is a prerequisite for investigating the function of CBS in SCD erythroblasts
So, Im going to assume the headline was written by some, a Nature editor or not, maybe the letters editor who wasnt paying enough attention
Instead, it acts as a downstream element within this growth hormone-IGF-1 axis
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12) (Simunkova et al
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