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Third, as we will detail below (see Potential Pathogenic Mechanisms of Hyperhomocysteinemia), our own work (Durand et al, 1997a) and the work of Lentz et al (1996) and Bellamy et al (1998) give experimental evidence that moderate hyperhomocysteinemia, which leads to vascular dysfunction, might increase the cardiovascular risk independently of other risk factors

Oct 2014;3(5):e001169

One such peptide, called PT-141 (a.k.a

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MASH: Metabolic dysfunction-associated steatohepatitis

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